Science Magazine | AAAS: Science Translational Medicine 中文 关注 脊髓少突胶质细胞的代谢重定位通过神经元Aβ42的产生驱动小鼠慢性疼痛 |科学转化医学 组织损伤重编程脊髓少突胶质细胞的代谢,改变髓鞘并触发神经元 Aβ42 产生,从而驱动疼痛慢性化。 Metabolic reallocation in spinal cord oligodendrocytes drives chronic pain in mice through neuronal Aβ42 production | Science Translational Medicine science.org Science Magazine | AAAS: Science Translational Medicine 中文 RSS thenote.app