Science Magazine | AAAS: Science Translational Medicine 中文 关注 针对CH25H以增强p62依赖的α-突触核蛋白自噬降解在帕金森病的细胞和小鼠模型中 | 科学转化医学 胆固醇 25-羟化酶与 p62 的竞争性结合抑制了α-突触核蛋白的自噬降解。 Targeting of CH25H to boost p62-dependent autophagic degradation of α-synuclein in cell and mouse models of Parkinson’s disease | Science Translational Medicine science.org
